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Integrated Sudden Cardiac Death Risk Prediction Model For Patients With Hypertrophic Cardiomyopathy.
Monda, Emanuele MD; Limongelli, Giuseppe MD, PhD
doi : 10.1161/CIRCULATIONAHA.122.063019
Volume 147(4) pgs. 281-360,e64-e69 January 24, 2023
Empagliflozin and Left Ventricular Remodeling in People Without Diabetes: Primary Results of the EMPA-HEART 2 CardioLink-7 Randomized Clinical Trial.
Connelly, Kim A. MBBS, PhD ,,,*; Mazer, C. David MD ,,,*; Puar, Pankaj MSc; Teoh, Hwee PhD; Wang, Chao-Hung MD, PhD; Mason, Tamique PhD; Akhavein, Farhad MD; Chang, Ching-Wen MD; Liu, Min-Hui RN, NP, PhD; Yang, Ning-I MB ChB; Chen, Wei-Siang MD; Juan, Yu-Hsiang MD; Opingari, Erika MSc; Salyani, Yaseen BSc; Barbour, William; Pasricha, Aryan; Ahmed, Shamon MD; Kosmopoulos, Andrew MSc; Verma, Raj; Moroney, Michael; Bakbak, Ehab HBSc; Krishnaraj, Aishwarya BScH; Bhatt, Deepak L. MD, MPH; Butler, Javed MD, MPH, MBA; Kosiborod, Mikhail N. MD; Lam, Carolyn S.P. MBBS, PhD; Hess, David A. PhD; Rizzi Coelho-Filho, Otavio MD, PhD, MPH; Lafreniere-Roula, Myriam BEng, PhD; Thorpe, Kevin E. MMath; Quan, Adrian MPhil; Leiter, Lawrence A. MD; Yan, Andrew T. MD; Verma, Subodh MD, PhD
doi : 10.1161/CIRCULATIONAHA.122.062769
Sodium-glucose cotransporter 2 inhibitors have been demonstrated to promote reverse cardiac remodeling in people with diabetes or heart failure. Although it has been theorized that sodium-glucose cotransporter 2 inhibitors might afford similar benefits in people without diabetes or prevalent heart failure, this has not been evaluated.
A Randomized Controlled Trial Comparing Apixaban With the Vitamin K Antagonist Phenprocoumon in Patients on Chronic Hemodialysis: The AXADIA-AFNET 8 Study.
Reinecke, Holger MD; Engelbertz, Christiane PhD; Bauersachs, Rupert MD; Breithardt, Gunter MD; Echterhoff, Hans-Herbert MD; Ger[latin sharp s], Joachim PhD; Haeusler, Karl Georg MD; Hewing, Bernd MD; Hoyer, Joachim MD; Juergensmeyer, Sabine PhD; Klingenheben, Thomas MD; Knapp, Guido PhD; Christian Rump, Lars MD; Schmidt-Guertler, Hans MD; Wanner, Christoph MD; Kirchhof, Paulus MD ,,,,*; Goerlich, Dennis PhD ,*
doi : 10.1161/CIRCULATIONAHA.122.062779
Non-vitamin K oral anticoagulants have become the standard therapy for preventing stroke and ischemic thromboembolism in most patients with atrial fibrillation (AF).
Effect of Intensive Blood Pressure Control on Troponin and Natriuretic Peptide Levels: Findings From SPRINT.
Berry, Jarett D. MD; Chen, Haiying MD, PhD; Nambi, Vijay MD, PhD; Ambrosius, Walter T. PhD; Ascher, Simon B. MD, MPH; Shlipak, Michael G. MD, MPH; Ix, Joachim H. MD, MAS; Gupta, Rajesh MD; Killeen, Anthony MD, PhD; Toto, Robert D. MD; Kitzman, Dalane W. MD; Ballantyne, Christie M. MD; de Lemos, James A. MD
doi : 10.1161/CIRCULATIONAHA.122.059960
Given the important role of cardiac injury and neurohormonal activation in the pathways leading from hypertension to heart failure and strong associations observed between hypertension and its sequelae on hs-cTnT (high-sensitivity cardiac troponin T) and NT-proBNP (N-terminal pro-B-type natriuretic peptide) levels, we hypothesized that intensive systolic blood pressure (SBP) lowering would decrease levels of hs-cTnT and NT-proBNP.
RNA-Binding Protein LIN28a Regulates New Myocyte Formation in the Heart Through Long Noncoding RNA-H19.
Rigaud, Vagner Oliveira Carvalho PhD; Hoy, Robert C. MS; Kurian, Justin MS; Zarka, Clare BS; Behanan, Michael BS; Brosious, Isabella BS; Pennise, Jennifer BS; Patel, Tej BS; Wang, Tao PhD; Johnson, Jaslyn PhD; Kraus, Lindsay M. PhD; Mohsin, Sadia PhD; Houser, Steven R. PhD; Khan, Mohsin PhD
doi : 10.1161/CIRCULATIONAHA.122.059346
Developmental cardiac tissue holds remarkable capacity to regenerate after injury and consists of regenerative mononuclear diploid cardiomyocytes. On maturation, mononuclear diploid cardiomyocytes become binucleated or polyploid and exit the cell cycle. Cardiomyocyte metabolism undergoes a profound shift that coincides with cessation of regeneration in the postnatal heart.
NLRP3 Inflammasome Activation Through Heart-Brain Interaction Initiates Cardiac Inflammation and Hypertrophy During Pressure Overload.
Higashikuni, Yasutomi MD, PhD; Liu, Wenhao MD; Numata, Genri MD, PhD; Tanaka, Kimie MD, PhD; Fukuda, Daiju MD, PhD; Tanaka, Yu MD, PhD; Hirata, Yoichiro MD, PhD; Imamura, Teruhiko MD, PhD; Takimoto, Eiki MD, PhD; Komuro, Issei MD, PhD; Sata, Masataka MD, PhD
doi : 10.1161/CIRCULATIONAHA.122.060860
Mechanical stress on the heart, such as high blood pressure, initiates inflammation and causes hypertrophic heart disease. However, the regulatory mechanism of inflammation and its role in the stressed heart remain unclear.
Interleukin-6 Receptor Polymorphism Attenuates Clonal Hematopoiesis-Mediated Coronary Artery Disease Risk Among 451 180 Individuals in the UK Biobank.
Vlasschaert, Caitlyn MD, MSc; Heimlich, J. Brett MD, PhD; Rauh, Michael J. MD, PhD; Natarajan, Pradeep MD, MMSc; Bick, Alexander G. MD, PhD
Letter by Halushka and Witwer Regarding Article, "Circulating MicroRNA-122-5p Is Associated With a Lack of Improvement in Left Ventricular Function After Transcatheter Aortic Valve Replacement and Regulates Viability of Cardiomyocytes Through Extracellular Vesicles".
Halushka, Marc K. MD, PhD; Witwer, Kenneth W. PhD
Letter by Li et al Regarding Article, "Circulating MicroRNA-122-5p Is Associated With a Lack of Improvement in Left Ventricular Function After Transcatheter Aortic Valve Replacement and Regulates Viability of Cardiomyocytes Through Extracellular Vesicles".
Li, Qi MD; Li, Zhuqing MD; Lu, Chengzhi PhD
Letter by Gutmann et al Regarding Article, "Circulating MicroRNA-122-5p Is Associated With a Lack of Improvement in Left Ventricular Function After Transcatheter Aortic Valve Replacement and Regulates Viability of Cardiomyocytes Through Extracellular Vesicles".
Gutmann, Clemens MD; Stojkovic, Stefan MD, PhD; Mayr, Manuel MD, PhD
