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Consequences of famine and malnourishment on kidney health
Gerjan NavisÂÂ
doi : 10.1038/s41581-023-00681-5
Volume 19 Issue 3, March 2023
Climate change-fuelled natural disasters and chronic kidney disease: a call for action
Amir Sapkota & Peter KotankoÂ
Heat and health inequity: acting on determinants of health to promote heat justice
Jeremy HessÂ
Environmental pollution, heat stress and kidney health: a need for integrated assessment
Alison P. Sanders & Nishad JayasundaraÂ
Disaster preparedness for patients with kidney disease
Jeffrey B. Kopp, Kenneth Lempert & Kristen FinneÂ
Informing clinical decision making during a pandemic � a call for better preparedness
Priya Vart, Luuk B. Hilbrands & Ron T. GansevoortÂ
Fasting induces structural and functional changes in the peritoneal membrane
Ellen F. CarneyÂÂ
The gut microbiome and hypertension
Joanne A. O’Donnell, Tenghao Zheng, Guillaume Meric & Francine Z. MarquesÂ
doi : 10.1038/s41581-022-00654-0
A large body of evidence has emerged in the past decade supporting a role for the gut microbiome in the regulation of blood pressure.
Youth-onset type 2 diabetes mellitus: an urgent challenge
Petter Bjornstad, Lily C. Chao, Melanie Cree-Green, Allison B. Dart, Malcolm King, Helen C. Looker, Dianna J. Magliano, Kristen J. Nadeau, Orit Pinhas-Hamiel, Amy S. Shah, Daniel H. van Raalte, Meda E. Pavkov & Robert G. NelsonÂ
doi : 10.1038/s41581-022-00645-1
The incidence and prevalence of youth-onset type 2 diabetes mellitus (T2DM) and its complications are increasing worldwide. Youth-onset T2DM has been reported in all racial and ethnic groups, but Indigenous peoples and people of colour are disproportionately affected.
Regulation of FGF23 production and phosphate metabolism by bone–kidney interactions
Rafiou Agoro & Kenneth E. WhiteÂ
Clinical practice recommendations for primary hyperoxaluria: an expert consensus statement from ERKNet and OxalEurope
Jaap W. Groothoff, Ella Metry, Lisa Deesker, Sander Garrelfs, Cecile Acquaviva, Reham Almardini, Bodo B. Beck, Olivia Boyer, Rimante Cerkauskiene, Pietro Manuel Ferraro, Luitzen A. Groen, Asheeta Gupta, Bertrand Knebelmann, Giorgia Mandrile, Shabbir S. Moochhala, Agnieszka Prytula, Jovana Putnik, Gill Rumsby, Neveen A. Soliman, Bhaskar Somani & Justine BacchettaÂ
doi : 10.1038/s41581-022-00661-1
Primary hyperoxaluria (PH) is an inherited disorder that results from the overproduction of endogenous oxalate, leading to recurrent kidney stones, nephrocalcinosis and eventually kidney failure; the subsequent storage of oxalate can cause life-threatening systemic disease.
