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A new Article Series for adipose tissue
doi : 10.1038/s41574-023-00832-5
Volume 19 Issue 5, May 2023
On the cutting edge: perspectives in bioenergetics
Melia Granath-Panelo, Anna Krook, Jared Rutter & Shingo KajimuraÂ
Interventions in people newly diagnosed with type 1 diabetes mellitus
Claire GreenhillÂ
Reprogramming lymph nodes in type 1 diabetes mellitus
Shimona StarlingÂÂ
CITED1 mediates sex-specific regulation of food intake in hypothalamic neurons
Olivia TysoeÂ
The metabolic benefits of exercise-induced hepatic autophagy
Claire GreenhillÂÂ
Engineering stem cells to recapitulate thyroid cancer
Shimona StarlingÂ
A novel resource to study endometriosis at the single-cell level
Luz GarcÃÂa-Alonso, Krina T. Zondervan & Roser Vento-TormoÂ
The gut microbiota in obesity and weight management: microbes as friends or foe?
Matthias Van Hul & Patrice D. CaniÂ
doi : 10.1038/s41574-022-00794-0
Obesity is caused by a long-term difference between energy intake and expenditure � an imbalance that is seemingly easily restored by increasing exercise and reducing caloric consumption. However, as simple as this solution appears, for many people, losing excess weight is difficult to achieve and even more difficult to maintain.
Metabolism and exercise: the skeletal muscle clock takes centre stage
Ryan A. Martin, Mark R. Viggars & Karyn A. EsserÂ
doi : 10.1038/s41574-023-00805-8
Circadian rhythms that influence mammalian homeostasis and overall health have received increasing interest over the past two decades. The molecular clock, which is present in almost every cell, drives circadian rhythms while being a cornerstone of physiological outcomes.
Intermuscular adipose tissue in metabolic disease
Bret H. Goodpaster, Bryan C. Bergman, Andrea M. Brennan & Lauren M. SparksÂ
doi : 10.1038/s41574-022-00784-2
Intermuscular adipose tissue (IMAT) is a distinct adipose depot described in early reports as a ‘fatty replacement’ or ‘muscle fat infiltration’ that was linked to ageing and neuromuscular disease.
Consensus Statement on the definition and classification of metabolic hyperferritinaemia
Luca Valenti, Elena Corradini, Leon A. Adams, Elmar Aigner, Saleh Alqahtani, Marco Arrese, Edouard Bardou-Jacquet, Elisabetta Bugianesi, Jose-Manuel Fernandez-Real, Domenico Girelli, Hannes Hagström, Benjamin Henninger, Kris Kowdley, Guido Ligabue, Donald McClain, Fabrice Lainé, Koji Miyanishi, Martina U. Muckenthaler, Alessia Pagani, Patrizia Pedrotti, Antonello Pietrangelo, Daniele Prati, John D. Ryan, Laura Silvestri, C. Wendy Spearman, Per StÃ¥l, Emmanuel A. Tsochatzis, Francesca Vinchi, Ming-Hua Zheng & Heinz ZollerÂ
doi : 10.1038/s41574-023-00807-6
Hyperferritinaemia is a common laboratory finding that is often associated with metabolic dysfunction and fatty liver. Metabolic hyperferritinaemia reflects alterations in iron metabolism that facilitate iron accumulation in the body and is associated with an increased risk of cardiometabolic and liver diseases.
