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BRAVE news: another one bites the dust
Jessica McHughÂÂ
doi : 10.1038/s41584-023-00957-x
Volume 19 Issue 5, May 2023
Allele-specific chromatin looping promotes IRF5-dependent autoimmunity
Robert PhillipsÂÂ
Mannosylation is a cause and target in lupus nephritis
Robert PhillipsÂÂ
Citrulline immunity in RA: CD8+ T cells enter the scene
Karine Chemin & Vivianne MalmströmÂ
The potential contribution of in silico studies to improved treatment of osteoarthritis
Corrinus C. van DonkelaarÂÂ
The 2022 ACR vaccination guideline: a call-to-action
Inés Colmegna & Michael LibmanÂ
Involvement of the secosteroid vitamin D in autoimmune rheumatic diseases and COVID-19
Maurizio Cutolo, Vanessa Smith, Sabrina Paolino & Emanuele GotelliÂ
doi : 10.1038/s41584-023-00944-2
Evidence supporting the extra-skeletal role of vitamin D in modulating immune responses is centred on the effects of its final metabolite, 1,25-dihydroxyvitamin D3 (1,25(OH)2D3, also known as calcitriol), which is regarded as a true steroid hormone.
Genetics and epigenetics of primary Sjögren syndrome: implications for future therapies
Gudny Ella Thorlacius, Albin Björk & Marie Wahren-HerleniusÂ
doi : 10.1038/s41584-023-00932-6
In primary Sjögren syndrome (pSS), chronic inflammation of exocrine glands results in tissue destruction and sicca symptoms, primarily of the mouth and eyes. Fatigue, arthralgia and myalgia are also common symptoms, whereas extraglandular manifestations that involve the respiratory, nervous and vascular systems occur in a subset of patients.
Mechanisms underlying the long-term and withdrawal effects of denosumab therapy on bone
Serge Ferrari & Bente LangdahlÂ
doi : 10.1038/s41584-023-00935-3
Denosumab, a human monoclonal antibody against receptor activator of nuclear factor-κB ligand (RANKL), is a potent inhibitor of osteoclast differentiation and activity.
Imbalance of threat and soothing systems in fibromyalgia: rephrasing an established mechanistic model?
Manuel MartÃÂnez-LavÃÂnÂÂ
Reply to ‘Imbalance of threat and soothing systems in fibromyalgia: rephrasing an established mechanistic model?’
Ana Margarida Pinto, Rinie Geenen, Tor D. Wager, Mark A. Lumley, Winfried Häuser, Eva Kosek, Jacob N. Ablin, Kirstine Amris, Jaime Branco, Dan Buskila, João Castelhano, Miguel Castelo-Branco, Leslie J. Crofford, Mary-Ann Fitzcharles, Marina López-Solà , Mariana LuÃÂs, Tiago Reis Marques, Philip J. Mease, Filipe Palavra, Jamie L. Rhudy, Lucina Q. Uddin, Paula Castilho, Johannes W. G. Jacobs & José A. P. da SilvaÂ
Hypothetical model ignores many important pathophysiologic mechanisms in fibromyalgia
Daniel J. Clauw, Ernest H. S. Choy, Vitaly Napadow, Anushka Soni, Kevin F. Boehnke, Bruce Naliboff, Afton L. Hassett, Anne Arewasikporn, Andrew Schrepf, Chelsea M. Kaplan, David Williams, Neil Basu, Rachel S. Bergmans, Richard E. Harris, Steven E. Harte, Andrea Chadwick & Gary J. MacfarlaneÂ
Reply to: Hypothetical model ignores many important pathophysiologic mechanisms in fibromyalgia
Ana Margarida Pinto, Rinie Geenen, Tor D. Wager, Winfried Häuser, Eva Kosek, Jacob N. Ablin, Kirstine Amris, Jaime Branco, Dan Buskila, João Castelhano, Miguel Castelo-Branco, Leslie J. Crofford, Mary-Ann Fitzcharles, Marina López-Solà , Mariana LuÃÂs, Tiago Reis Marques, Philip J. Mease, Filipe Palavra, Jamie L. Rhudy, Lucina Q. Uddin, Paula Castilho, Johannes W. G. Jacobs & José A. P. da SilvaÂ
