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Intersectionality as a lens for achieving kidney health justice
Elle Lett, Carmen H. Logie & Dinushika MohottigeÂ
doi : 10.1038/s41581-023-00715-y
Volume 19 Issue 6, June 2023
Including sexual orientation and gender identity data to advance nephrology care
Ken Sutha & Carl G. Streed JrÂ
Sexual orientation and gender identity in organ and tissue donation and transplantation
Murdoch Leeies, Emily Christie & David CollisterÂ
Interim analysis indicates sparsentan PROTECTs in IgAN
Susan J. AllisonÂÂ
Mannose as a driver of γδ T cell kidney infiltration
Monica WangÂ
Considerations in the care of transgender persons
Sofia B. Ahmed, Lauren B. Beach, Joshua D. Safer, Jaimie F. Veale & Cameron T. WhitleyÂ
The role of lysosomes in metabolic and autoimmune diseases
Frédéric Gros & Sylviane MullerÂ
doi : 10.1038/s41581-023-00692-2
Lysosomes are catabolic organelles that contribute to the degradation of intracellular constituents through autophagy and of extracellular components through endocytosis, phagocytosis and macropinocytosis. They also have roles in secretory mechanisms, the generation of extracellular vesicles and certain cell death pathways.
The pathophysiology of distal renal tubular acidosis
Carsten A. Wagner, Robert Unwin, Sergio C. Lopez-Garcia, Robert Kleta, Detlef Bockenhauer & Stephen WalshÂ
doi : 10.1038/s41581-023-00699-9
The kidneys have a central role in the control of acid–base homeostasis owing to bicarbonate reabsorption and production of ammonia and ammonium in the proximal tubule and active acid secretion along the collecting duct. Impaired acid excretion by the collecting duct system causes distal renal tubular acidosis (dRTA), which is characterized by the failure to acidify urine below pH 5.5.
Sepsis-associated acute kidney injury: consensus report of the 28th Acute Disease Quality Initiative workgroup
Alexander Zarbock, Mitra K. Nadim, Peter Pickkers, Hernando Gomez, Samira Bell, Michael Joannidis, Kianoush Kashani, Jay L. Koyner, Neesh Pannu, Melanie Meersch, Thiago Reis, Thomas Rimmelé, Sean M. Bagshaw, Rinaldo Bellomo, Vicenzo Cantaluppi, Akash Deep, Silvia De Rosa, Xose Perez-Fernandez, Faeq Husain-Syed, Sandra L. Kane-Gill, Yvelynne Kelly, Ravindra L. Mehta, Patrick T. Murray, Marlies Ostermann, John Prowle, Zaccaria Ricci, Emily J. See, Antoine Schneider, Danielle E. Soranno, Ashita Tolwani, Gianluca Villa, Claudio Ronco & Lui G. ForniÂ
doi : 10.1038/s41581-023-00683-3
Sepsis-associated acute kidney injury (SA-AKI) is common in critically ill patients and is strongly associated with adverse outcomes, including an increased risk of chronic kidney disease, cardiovascular events and death. The pathophysiology of SA-AKI remains elusive, although microcirculatory dysfunction, cellular metabolic reprogramming and dysregulated inflammatory responses have been implicated in preclinical studies.
