SWI is very sensitive to slow-flow vascular malformations such as CCM, as in this child with a large left temporal CCM (dashed arrows), with a characteristic "popcorn" appearance on CT and MRI. There are typical internal calcifications on CT (A), hyperintense subacute blood products on T1-weighted imaging (B), mild internal enhancement on postcontrast T1-weighted imaging (C), and peripheral hypointense hemosiderin on FLAIR (D) and T2-weighted imaging (E). The lesion appears larger on SWI (F) due to blooming artifact. More important, additional smaller CCMs are detected (arrows), which indicate that the patient likely has multiple familial CCM syndrome (which can occur in as many as one-third of patients with CCM). SWI parameters are: 3 Tesla, TE = 20 milliseconds, TR = 29 milliseconds, FA = 15°, field of vision 250 mm × 188 mm, matrix 448 × 336, 2 mm thick acquisition displayed with 16 mm minimum intensity projection.
From: Liu C, Li W, Tong KA, et al. Susceptibility-weighted imaging and quantitative susceptibility mapping in the brain. J Magn Reson Imaging 2015; 42(1):23-41.
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