Version May 2024
| Decreased potassium intake |
| Increased entry into cells |
| An elevation in extracellular pH |
| Increased availability of insulin |
| Elevated β-adrenergic activity - stress or administration of beta agonists |
| Hypokalemic periodic paralysis |
| Marked increase in blood cell production |
| Hypothermia |
| Chloroquine intoxication |
| Increased gastrointestinal losses |
| Vomiting |
| Diarrhea |
| Tube drainage |
| Laxative abuse |
| Increased urinary losses |
| Diuretics |
| Primary mineralocorticoid excess |
| Loss of gastric secretions |
| Nonreabsorbable anions |
| Renal tubular acidosis |
| Hypomagnesemia |
| Amphotericin B |
| Salt-wasting nephropathies - including Bartter's or Gitelman's syndrome |
| Polyuria |
| Increased sweat losses |
| Dialysis |
| Plasmapheresis |
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