Version May 2024
| Acute phosphate load |
| Endogenous |
| Cell lysis (tumor lysis syndrome, rhabdomyolysis) |
| Exogenous |
| Phosphate-containing medications (laxatives, fosphenytoin) |
| Intestinal uptake (vitamin D toxicity) |
| Cellular shift |
| Lactic or ketoacidosis |
| Decreased renal clearance |
| Reduced glomerular filtration rate |
| Acute kidney injury |
| Chronic kidney disease |
| Increased tubular reabsorption |
| Hypoparathyroidism or pseudohypoparathyroidism |
| Acromegaly |
| Bisphosphonates |
| Cinacalcet |
| Vitamin D toxicity (also increases intestinal absorption) |
| Familial tumoral calcinosis |
| Fibroblast growth factor receptor inhibitors |
| Pseudohyperphosphatemia |
| Endogenous |
| Hyperglobulinemia (multiple myeloma, Waldenström macroglobulinemia) |
| Hyperlipidemia |
| Hemolysis |
| Hyperbilirubinemia |
| Exogenous |
| Medications (amphotericin B, heparin, tissue plasminogen activator) |
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